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Vestibular disorders: BPPV, vestibular neuritis, Ménière's, third window, bilateral loss and central signs

Most disorders are told apart by how long the vertigo lasts and what triggers it: seconds on changing position — BPPV; minutes to hours in attacks with ear symptoms — Ménière's; days of constant vertigo — vestibular neuritis (or stroke, picked out with HINTS). Third window and bilateral loss are patterns recognised from test findings.

Why it matters in the exam

BPPV: otoconia shed from the utricle enter a canal, most often the posterior one. Brief vertigo (under a minute) on position changes; the Dix-Hallpike test shows, after a short latency, upbeating torsional nystagmus that fatigues. Treatment: a canalith repositioning manoeuvre (Epley), with no drugs and no positional restrictions afterwards. For the horizontal canal the supine roll test is used: geotropic nystagmus to both sides means free particles (canalithiasis), and the side with the stronger response is the sick one. Vestibular neuritis: continuous spinning vertigo for days, nausea, no hearing loss; direction-fixed nystagmus beating away from the affected side, and a positive head impulse toward the affected side. When only the superior nerve is hit, the cVEMP (saccule, inferior nerve) is spared.

Central signs: by HINTS, in an acute vestibular syndrome — a normal head impulse, direction-changing nystagmus and a vertical eye misalignment (skew) point to stroke. Pure downbeating nystagmus with no latency and no fatigue is central too. Ménière's: spontaneous vertigo attacks of 20 minutes to 12 hours with low-frequency loss, tinnitus and fullness. Third window (superior canal dehiscence): a low-frequency air-bone gap with normal tympanogram and reflexes, better-than-normal bone conduction, hearing one's own body sounds and vertigo from loud sounds; a large VEMP with a low threshold; an enlarged vestibular aqueduct also gives a high VEMP. Cholesteatoma can erode the horizontal canal and create a fistula — vertigo with pressure on the ear. Bilateral loss (for example after gentamicin): oscillopsia — the world "jumps" when walking — and unsteadiness in the dark; rehabilitation stresses substitution (vision and sensation from the feet), because there is nothing left to adapt. In one-sided loss gaze-stabilisation exercises (adaptation) are stressed.

Key points

Summary table

DisorderPresentationTest or treatment
Posterior canal BPPVSeconds, on position changeDix-Hallpike: upbeating and torsional, fatigues; Epley
Horizontal canal BPPVLying down and rolling the headGeotropic — the affected side is the stronger
Vestibular neuritisDays, constant, no hearing lossNystagmus away from the affected side; positive head impulse
Stroke (central)Acute syndromeHINTS: normal head impulse, direction-changing nystagmus, skew
Ménière's20 minutes to 12 hours, spontaneousLow-frequency loss, tinnitus, fullness
Third windowLow-frequency gap, normal tympanogram and reflexesLarge VEMP; CT
Bilateral lossOscillopsia, unsteadiness in the darkRehabilitation by substitution

A free exam question

On the supine roll test, horizontal geotropic nystagmus (beating toward the ground) appears on both sides, stronger and longer when the head is turned to the right. What is the most likely diagnosis?

  1. A. Left horizontal canal BPPV, canalithiasis type
  2. B. Right horizontal canal BPPV, cupulolithiasis type
  3. C. Right posterior canal BPPV
  4. D. Right horizontal canal BPPV, canalithiasis type

See the answer and explanation

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Reviewed by the Examqo AI Professor · 2026-11-02. The text may contain errors; when in doubt, check the source.